Source: Chemico-Biological Interactions. Unidade: ICB
Assunto: PARASITOLOGIA
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SOLETTI, Rossana C. et al. Peptide gomesin triggers cell death through L-type channel calcium influx, MAPK/ERK, PKC and PI3K signaling and generation of reactive oxygen species. Chemico-Biological Interactions, v. 186, n. 2, p. 135-143, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2010.04.012. Acesso em: 05 maio 2024.APA
Soletti, R. C., Barrio, L. del, Daffre, S., Miranda, A., Borges, H. L., Moura-Neto, V., et al. (2010). Peptide gomesin triggers cell death through L-type channel calcium influx, MAPK/ERK, PKC and PI3K signaling and generation of reactive oxygen species. Chemico-Biological Interactions, 186( 2), 135-143. doi:10.1016/j.cbi.2010.04.012NLM
Soletti RC, Barrio L del, Daffre S, Miranda A, Borges HL, Moura-Neto V, Lopez MG, Gabilan NH. Peptide gomesin triggers cell death through L-type channel calcium influx, MAPK/ERK, PKC and PI3K signaling and generation of reactive oxygen species [Internet]. Chemico-Biological Interactions. 2010 ; 186( 2): 135-143.[citado 2024 maio 05 ] Available from: https://doi.org/10.1016/j.cbi.2010.04.012Vancouver
Soletti RC, Barrio L del, Daffre S, Miranda A, Borges HL, Moura-Neto V, Lopez MG, Gabilan NH. Peptide gomesin triggers cell death through L-type channel calcium influx, MAPK/ERK, PKC and PI3K signaling and generation of reactive oxygen species [Internet]. Chemico-Biological Interactions. 2010 ; 186( 2): 135-143.[citado 2024 maio 05 ] Available from: https://doi.org/10.1016/j.cbi.2010.04.012
